Abstract Several recent studies have suggested that antitaxial fibrous veins may form without fracturing, and not by the commonly invoked crack-seal mechanism. It has also been suggested that such veins would derive their nutrients locally by diffusional transport. This hypothesis was tested on carbonaceous shale-hosted antitaxial fibrous calcite veins from Oppaminda Creek in the northern Flinders Ranges, South Australia. Apart from their fibrous texture, these veins lack the classical features of crack-seal veins, such as wallrock-parallel inclusion bands. Diffusional transport of locally derived calcite cannot explain all major and trace element data of the veins and their adjacent wallrock and indicate that part of the calcite was transported over distances of at least >decimetres, probably ≫100m. Sr isotopic fingerprinting shows that an external fluid that carried radiogenic Sr must have percolated through the system. Fluid flow was pervasive as there is no evidence that this fluid preferentially percolated through the veins. Our data support the view that antitaxial fibrous veins of the type found at Oppaminda Creek grew in the absence of fractures, but show that such veins do not necessarily indicate local diffusional transport. Our data confirm a recently postulated basin-wide fluid flow event around 586 Ma that is probably related to copper mineralization in the area.

Two models for the heating responsible for granite generation during convergent deformation may be distinguished on the basis of the length- and time-scales associated with the thermal perturbation, namely: (1) long-lived, lithospheric-scale heating as a conductive response to the deformation, and (2) transient, localised heating as a response to advective heat sources such as mantle-derived melts. The strong temperature dependence of lithospheric rheology implies that the heat advected within rising granites may affect the distribution and rates of deformation within the developing orogen in a way that reflects the thermal regime attendant on granite formation; this contention is supported by numerical models of lithospheric deformation based on the thin-sheet approximation. The model results are compared with geological and isotopic constraints on granite genesis in the southern Adelaide Fold Belt where intrusion spans a 25 Ma convergent deformation cycle, from about 516 to 490 Ma, resulting in crustal thickening to 50–55 km. High-T metamorphism in this belt is spatially restricted to an axis of magmatic activity where the intensity and complexity of deformation is significantly greater, and may have started earlier, than in adjacent low-grade areas. The implication is that granite generation and emplacement is a causative factor in localising deformation, and on the basis of the results of the mechanical models suggests that granite formation occurred in response to localised, transient crustal heating by mantle melts. This is consistent with the Nd- and Sr-isotopic composition of the granites which seems to reflect mixed sources with components derived both from the depleted contemporary mantle and the older crust.